BOUAOUICHE ABDERRAHMENE and BOULAKOUD MOHAMED SALAH (2017) STUDY AND MELANOCYTE ADRENOCORTICOTROPIC EFFECTS ON SUGAR METABOLISM AND IMMUNE RESPONSE IN RABBITS, ORYCTOLAGUS CUNICULUS. Best Journals : International Journal of Humanities, Arts, Medicine and Sciences (BEST: IJHAMS) , 4(10), 109-120, Thomson Reuters' RESEARCHERID indexed http://www.researcherid.c om/rid/F-9906-2015
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Abstract
The functioning of the pineal gland, the transducer body of environmental information to the neuroendocrine
system was subjected to a circadian rhythm.
Melatonin is the main neuro-hormone expressing this operation. It is synthesized in the pinealocytes after
conversion of serotonin via N-acetyl-transferase enzyme, the same subject to a photoperiodic modulation (activation of
dark inhibition by light). Some scientists have suggested that melatonin is involved in diabetic disease and which expresses
have a diabetogenic effect. To this study the effect of this hormone on glucose metabolism has long been subject to
controversy agreeing in effect and hyperinsulinemic hypoglycaemic effect. In order to illustrate the level of interaction of
melatonin with neuro-immune- corticotrophin axis and its impact on carbohydrate metabolism, we studied the impact
homeostatic (glucose) through the solicitation of two control systems (gland pineal and corticotrophin axis). We and found
that melatonin could have an indirect influence on insulin control (glucose metabolism) to the levels of the growth
hormone axis (somatostatin) and adrenocorticotropic (corticotrophin). In addition, we have suggested that melatonin might
limit the hyperglycaemic action of corticosteroids by direct action at peripheral level.
KEYWORDS: Pineal Gland, Melatonin, Neuro-Immuno-Corticotrop
system was subjected to a circadian rhythm.
Melatonin is the main neuro-hormone expressing this operation. It is synthesized in the pinealocytes after
conversion of serotonin via N-acetyl-transferase enzyme, the same subject to a photoperiodic modulation (activation of
dark inhibition by light). Some scientists have suggested that melatonin is involved in diabetic disease and which expresses
have a diabetogenic effect. To this study the effect of this hormone on glucose metabolism has long been subject to
controversy agreeing in effect and hyperinsulinemic hypoglycaemic effect. In order to illustrate the level of interaction of
melatonin with neuro-immune- corticotrophin axis and its impact on carbohydrate metabolism, we studied the impact
homeostatic (glucose) through the solicitation of two control systems (gland pineal and corticotrophin axis). We and found
that melatonin could have an indirect influence on insulin control (glucose metabolism) to the levels of the growth
hormone axis (somatostatin) and adrenocorticotropic (corticotrophin). In addition, we have suggested that melatonin might
limit the hyperglycaemic action of corticosteroids by direct action at peripheral level.
KEYWORDS: Pineal Gland, Melatonin, Neuro-Immuno-Corticotrop
Information
| Item Type | Journal |
|---|---|
| Divisions |
» Laboratory for Terrestrial and Aquatic Ecosystems |
| ePrint ID | 683 |
| Date Deposited | 2017-03-03 |
| Further Information | Google Scholar |
| URI | https://univ-soukahras.dz/en/publication/article/683 |
BibTex
@article{uniusa683,
title={STUDY AND MELANOCYTE ADRENOCORTICOTROPIC EFFECTS ON SUGAR METABOLISM AND IMMUNE RESPONSE IN RABBITS, ORYCTOLAGUS CUNICULUS},
author={BOUAOUICHE ABDERRAHMENE and BOULAKOUD MOHAMED SALAH},
journal={Best Journals : International Journal of Humanities, Arts, Medicine and Sciences (BEST: IJHAMS)}
year={2017},
volume={4},
number={10},
pages={109-120},
publisher={Thomson Reuters' RESEARCHERID indexed http://www.researcherid.c om/rid/F-9906-2015}
}
title={STUDY AND MELANOCYTE ADRENOCORTICOTROPIC EFFECTS ON SUGAR METABOLISM AND IMMUNE RESPONSE IN RABBITS, ORYCTOLAGUS CUNICULUS},
author={BOUAOUICHE ABDERRAHMENE and BOULAKOUD MOHAMED SALAH},
journal={Best Journals : International Journal of Humanities, Arts, Medicine and Sciences (BEST: IJHAMS)}
year={2017},
volume={4},
number={10},
pages={109-120},
publisher={Thomson Reuters' RESEARCHERID indexed http://www.researcherid.c om/rid/F-9906-2015}
}